Publications

• A Retrospective Study of Chronic Post-Surgical Pain following Thoracic Surgery: Prevalence, Risk Factors, Incidence of Neuropathic Component, and Impact on Qualify of Life
  Peng, Z., Li, Hl., Zhang, C., Qian, X., Feng, Z., & Zhu, S. (2014). A Retrospective Study of Chronic Post-Surgical Pain following Thoracic Surgery: Prevalence, Risk Factors, Incidence of Neuropathic Component, and Impact on Qualify of Life. PLOS ONE, 9(2).
• CT-Guided Percutaneous Infrazygomatic Radiofrequency Neurolysis Through Foramen Rotundum to Treat V2 Trigeminal Neuralgia
  Huang, B., Yao, M., Feng, Z., Guo, J., Zereshki, A., & Qian* Xiang. (2014). CT-Guided Percutaneous Infrazygomatic Radiofrequency Neurolysis Through Foramen Rotundum to Treat V2 Trigeminal Neuralgia. Pain Medicine, (April 9).
• Optical control of neuronal excitation and inhibition using a single opsin protein, ChR2
  Liske, H., Qian, X., Anikeeva, P., Deisseroth, K., & Delp, S. (2013). Optical control of neuronal excitation and inhibition using a single opsin protein, ChR2. SCIENTIFIC REPORTS, 3.
• Deep Vein Thrombosis and Pulmonary Embolism Cosmin, G., & Qian, X. (2011). Deep Vein Thrombosis and Pulmonary Embolism (Vol. 470).
  
• G protein-activated inwardly rectifying potassium channels mediate depotentiation of long-term potentiation Chung, H. J., Ge, W.-P., Qian, X., Wiser, O., Jan, Y. N., & Jan, L. Y. (2009). G protein-activated inwardly rectifying potassium channels mediate depotentiation of long-term potentiation. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 106(2), 635-640.
  
• Neuronal activity regulates phosphorylation-dependent surface delivery of G protein-activated inwardly rectifying potassium channels Chung, H. J., Qian, X., Ehlers, M., Jan, Y. N., & Jan, L. Y. (2009). Neuronal activity regulates phosphorylation-dependent surface delivery of G protein-activated inwardly rectifying potassium channels. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 106(2), 629-634.
  
• SK channels mediate NADPH oxidase-independent reactive oxygen species production and apoptosis in granulocytes Fay, A. J., Qian, X., Jan, Y. N., & Jan, L. Y. (2006). SK channels mediate NADPH oxidase-independent reactive oxygen species production and apoptosis in granulocytes. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 103(46), 17548-17553.
  
• Intra- and intersubunit cooperativity in activation of BK channels by Ca2+ (vol 128, pg 389, 2006) Qian, X., Niu, X., & Magleby, K. L. (2006). Intra- and intersubunit cooperativity in activation of BK channels by Ca2+ (vol 128, pg 389, 2006). JOURNAL OF GENERAL PHYSIOLOGY, 128(5), 629-629.
  
• K+ channel selectivity depends on kinetic as well as thermodynamic factors Grabe, M., Bichet, D., Qian, X., Jan, Y. N., & Jan, L. Y. (2006). K+ channel selectivity depends on kinetic as well as thermodynamic factors. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 103(39), 14361-14366.
  
• Modulation of basal and receptor-induced GIRK potassium channel activity and neuronal excitability by the mammalian PINS homolog LGN Wiser, O., Qian, X., Ehlers, M., Ja, W. W., Roberts, R. W., & Jan, L. Y. (2006). Modulation of basal and receptor-induced GIRK potassium channel activity and neuronal excitability by the mammalian PINS homolog LGN. NEURON, 50(4), 561-573.
  
• Linker-gating ring complex as passive spring and Ca2+-dependent machine for a voltage-and Ca2+-activated potassium channel (vol 42, pg 745, 2004) Niu, X. W., Qian, X., & Magleby, K. L. (2005). Linker-gating ring complex as passive spring and Ca2+-dependent machine for a voltage-and Ca2+-activated potassium channel (vol 42, pg 745, 2004). NEURON, 45(4), 637-637.
  
• beta 1 subunits facilitate gating of BK channels by acting through the Ca2+, but not the Mg2+, activiating mechanisms Qian, X., & Magleby, K. L. (2003). beta 1 subunits facilitate gating of BK channels by acting through the Ca2+, but not the Mg2+, activiating mechanisms. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 100(17), 10061-10066.
  
• Slo1 tail domains, but not the Ca2+ bowl, are required for the beta 1 subunit to increase the apparent Ca2+ sensitivity of BK channels Qian, X., Nimigean, C. M., Niu, X. W., Moss, B. L., & Magleby, K. L. (2002). Slo1 tail domains, but not the Ca2+ bowl, are required for the beta 1 subunit to increase the apparent Ca2+ sensitivity of BK channels. JOURNAL OF GENERAL PHYSIOLOGY, 120(6), 829-843.